Early diagnosis, treatment and aftercare using Bicom bioresonance therapy

Ingo E. Bauer, Naturopath, Speyer

INTRODUCTION

Dear colleagues,
This year, as before, I am pleased to welcome a number of new colleagues who have attended my seminars.
The topic cerebral apoplexy or stroke is one which weighs on the minds of many of our patients.
Whether through fear of suffering a stroke themselves or the experience of a family member having suffered one.
I should like here to examine once again in more detail the causes, current state of knowledge and experiences of our centre for diagnosis and therapy.
Please forgive me if I first examine the causes. As our auditorium contains such a mixture, doctors, nonmedical practitioners, vets, dentists and naturopathic vets, I should like to bring everyone up to date.

WHAT IS A STROKE?

A stroke is the result of a sudden circulatory disturbance in the brain. As a result, the nerve cells in the brain receive insufficient oxygen and nutrients and they die.
Apoplexy, brain hemorrhage or cerebra infarction are other terms.
Theoretically anyone young or old can suffer a stroke. One in two sufferers is of working age, approx. 5 % are even under 40.
Apoplexy is the third most common cause of death in Germany. It affects 20 % of over 65s. Mortality is around 10 %.

HOW DOES A STROKE DEVELOP?

a) through inadequate circulation (ischaemia) because a blood vessel leading to the brain is suddenly blocked or

b) through a haemorrhage when a blood vessel suddenly ruptures.

Restricted circulation to the brain is by far the most common cause of strokes. It can occur in var-ious ways.

Thrombotic ischaemia

Here an artery is obstructed by a blood clot (thrombus) which has formed in a larger or smaller blood vessel in the brain.
These vascular occlusions do not usually occur in healthy cerebral vessels but in those whose walls are damaged by arteriosclerosis.

Embolic ischaemia

This starts with blood clots which have formed in the heart or in the large vessels leading to the brain, e. g. carotid artery.
Material can become detached from these blood clots and be carried by the blood stream to the brain where they obstruct an important blood vessel.
Around 15 % of strokes arise through what is known as vascular rupture.

Rupture of a blood vessel

TYPES OF APOPLEXY

Transitory ischaemic attacks (TIA)

This describes a neurological deficit lasting a few minutes, rarely longer than 1 to 2 hours. By defi-nition, it is limited to 24 hours.
Symptoms:

• impaired balance with or without dizziness

• loss of vision and hearing

• impaired speech or understanding of the spoken word

• inability to read, count or write

• numbness in arms or legs and bending in of the legs

Prolonged reversible ischaemic neurological deficit (PRIND)

This is a delayed circulatory disturbance of the brain. It develops over the course of 24 to 28 hours.
The symptoms are similar to those of a TIA. Regression can take up to 3 weeks.

Stroke in evolution

This can last hours or even days. During this time the level of neurological deficit increases. Oede-mas frequently form leading to changes in the pa-tient’s state of consciousness and even uncon-sciousness.
The symptoms are only partially reversible.

Completed stroke

Here neurological disturbance occurs in its full in-tensity.
The symptoms are serious and do not recede or if so, only to a limited extent.

Intracerebral haemorrhage

The causes are frequently micro-aneurysms, tumours or blood coagulation disease.
The symptoms are determined by the site of the haemorrhage and its severity.

Subarachnoid haemorrhage

This is usually caused by a congenital or acquired aneurysm.
It can occur at any age, although it is particu-larly common in young or middle age.
It makes up 5 % of cases.

Subdural haemorrhage

This is generally caused by a trauma, by an accident or a fall.

RISKS

• High blood pressure

• smoking

• bradyarrhythmia

• diabetes

• high cholesterol level

• overweight

• lack of exercise

• familial disposition.

According to the latest research from Boston, USA, also Chlamydia pneumoniae and Helicobacter pylori.
The head clinic in Heidelberg is conducting a detailed investigation into genetic causes.

OUR EXPERIENCE

Here in Speyer we conduct a large number of vital blood tests. I should now like to show you a couple of examples which clearly demonstrate the problems and prophylactic possibilities associated with strokes.

Example of patient: K. G., born 12.08.46

Case history

Date of admission 16.08.02. Stroke six months earlier with right-side paralysis. Current situation: paralysis of the right arm, severe restriction in use of the right leg.

Therapy

As with all our patients, we first have their stools tested for any healthy intestinal bacteria, pathogenic bacteria, yeast fungi and moulds present. With the following result: no evidence of physiological bacterial flora, pH-value: 5.8 (norm 6.2-6.8).
When he took the physiological bacteria, he complained of pain in his paralysed arm. Would you believe it! In the arm in which he usually has no sensation.
We had to reduce the dose which brought this back to normal.
After building up the intestines, we always test the vital blood using dark ground illumination. Yet you can still see the changes in the blood cells with a normal bright-field microscope.
Let this lecture motivate you to check what I have said on your patients. Dust off your micro-scopes and try it out for yourselves.
Our next step was to restore the blood’s opti-mal flow properties.
For this we took the patient’s capillary blood and applied the following programs:

• 430 liver detoxication

• 970 toxin elimination

• 480 renal activation.

The first series of therapy consisted of 3 treat-ment sessions at weekly intervals.
After the first session, Herr G. told me enthu-siastically that the pain in his arm was receding and a pleasant tingling had set in.
After the second session he could tell without looking whether his hand was clenched, closed or open.
After the third session he could open his clench-ed hand himself in a controlled manner.
I should just like to say here that we had not yet done anything to stimulate the nerves or regen-erate the cells.
These therapies followed

• cell regeneration

• cell stimulation

• nerves chronic/acute

• basic therapy

once a week in each case.
The first session resulted in pain in the legs, provoking complaints from the patient!
Following a serious discussion, I was able to persuade him that this was a good sign.
During the week following treatment the pa-tient began organising his forthcoming move. He packed cases and sorted them. His wife, who told me this, was very pleased.
By concentrating on this job, he completely forgot his zimmer frame.
Now that he has moved to another town we have lost contact with him at the moment but I should be glad to report on the continued healing process at one of the next congresses.

PREVENTION

Dear colleagues, please don’t rely on the custom-ary blood counts. If you still have a microscope available in your practice, carry out your own count with your patient’s blood. You don’t need a dark ground microscope, a normal microscope will also show you where the problem lies.
Traditional blood counts only give us evidence of how many of the individual blood cells are present. Looking through the microscope also shows us whether the blood cells are also capable of doing their job.
Here are some examples:

You see, erythrocyte conglobation is also visible with a normal microscope.
And this is also the first point in the matter of prevention. If I discover a family disposition from the patient’s case history, the microscope is my first port of call.
And then: have the lab check the stools and also the presence of healthy bacteria.
After building up a healthy intestinal flora, the blood is tested using dark ground microscopy.

Normal blood count

Blood count one day after Frankfurt marathon

Blood count with protein clumping

Erythrocyte rolls forming

Erythrocyte rolls formation

Erythrocyte rolls formation

Uric-acid crystal

Uric-acid crystal

Depending upon the result, the blood’s flow properties are restored with the programs described above (430, 970, 480).
Once this is achieved, the patient has a checkup six months later. If this is normal, the next check-up is scheduled for one year later.
The need to reduce the risks must be repeatedly emphasized.
Obviously acute cases should first be treated with the usual intensive medical care and then fol-lowing the procedure described in the example.

• creating the conditions for BICOM optima®l care of the body

• creating BICOM optima®l flow properties for the blood

• stimulating cell regeneration

• monitoring through routine examinations.

AFTERCARE

This consists of regular follow-up examinations.

CONCLUDING REMARKS

o, dear colleagues! I hope I have been able to encourage you to dig out and dust off your micro-scopes and do a bit of research for the good of your patients.
You will soon be able to consult this lecture and the video sequences on the interne by visiting my home page at www.bauer-net.de.
This lecture will also be available as a CD- ROM presentation which will run automatically on any computer.
If you have any questions or suggestions, please get in touch with me. (Refer to the Annex at the end of this volume.)